Difference between revisions of "Talk:20.109(S17):Module 2"
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*[[Media:Nagel et al PNAS 2014.pdf| Multiplexed DNA repair assays for multiple lesions and multiple doses via transcription inhibition and transcriptional mutagenesis.]] | *[[Media:Nagel et al PNAS 2014.pdf| Multiplexed DNA repair assays for multiple lesions and multiple doses via transcription inhibition and transcriptional mutagenesis.]] | ||
*[[Media:Treszezamsky et al CanRes 2007.pdf| BRCA1- and BRCA2-Deficient Cells Are Sensitive to Etoposide-Induced DNA Double-Strand Breaks via Topoisomerase II.]] | *[[Media:Treszezamsky et al CanRes 2007.pdf| BRCA1- and BRCA2-Deficient Cells Are Sensitive to Etoposide-Induced DNA Double-Strand Breaks via Topoisomerase II.]] | ||
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| + | ==quantitative PCR data== | ||
==CellTiter Glo luminescence cell viability assay== | ==CellTiter Glo luminescence cell viability assay== | ||
Revision as of 19:39, 13 April 2017
Contents
References from Prof. Samson Lecture 8
- A syngeneic variance library for functional annotation of human variation: application to BRCA2.
- Hypersensitivity of Nonhomologous DNA End-joining Mutants to VP-16 and ICRF-193.
- Compound screening in BRCA2 (–/–) GeneArt Engineered Cell Model.
- p63 and p73 Transcriptionally Regulate Genes Involved in DNA Repair.
- Multiplexed DNA repair assays for multiple lesions and multiple doses via transcription inhibition and transcriptional mutagenesis.
- BRCA1- and BRCA2-Deficient Cells Are Sensitive to Etoposide-Induced DNA Double-Strand Breaks via Topoisomerase II.
quantitative PCR data
CellTiter Glo luminescence cell viability assay
Western blot images
Instructor's Western Blot
T/R Western Blots
W/F Western Blots
